Liu L, Liu S, Shen L, et al. - Researchers analyzed a large Chinese cohort of patients with hypertrophic obstructive cardiomyopathy (HOCM) to examine the links between the development of nonsustained ventricular tachycardia (NSVT) and circulating biomarkers representing myocardial injury (cardiac troponin I, cTnI), cardiac function (N‐terminal pro‐brain natriuretic peptide), and inflammation (high‐sensitivity C‐reactive protein). This study involved 755 consecutive HOCM patients. Two groups were defined based on 24‐hour Holter monitoring: NSVT group (n = 138) and the nonventricular tachycardia (non‐VT) group (n = 617). A progressive rise in cTnI levels was evident not only with the aggravation of ventricular arrhythmic events, but also with the increasing risk of sudden cardiac death (SCD). Overall, raised cTnI was identified as an independent determinant of NSVT, and it appeared to be useful for evaluating the clinical status of ventricular arrhythmias as well as the risk of SCD among patients experiencing HOCM.